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Assessment of sodium butyrate’s pharmacological effects on gentamicin-induced nephrotic disorders in a rat model
Publication details: New York SAGE 2024Edition: Vol.15(4), DecDescription: 417–426pOnline resources: In: Journal of pharmacology and pharmacotherapeuticsSummary: Sodium butyrate (SB) used to increase oxidative stress was reduced and the glutathione redox pathway was boosted. It inhibits proliferation, induction of differentiation, and induction or repression of gene expression and it has also anti-inflammatory action. This is endorsed for suppression of nephrotic symptoms.| Item type | Current library | Status | Barcode | |
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School of Pharmacy Archieval Section | Not for loan | 2025-0275 |
Total holds: 0
Sodium butyrate (SB) used to increase oxidative stress was reduced and the glutathione redox pathway was boosted. It inhibits proliferation, induction of differentiation, and induction or repression of gene expression and it has also anti-inflammatory action. This is endorsed for suppression of nephrotic symptoms.
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